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Acute respiratory failure, type II, is characterized by the inability of the respiratory system to maintain adequate gas exchange, leading to hypercapnia (elevated carbon dioxide levels in the blood) and often accompanied by hypoxemia (low blood oxygen levels). This condition typically arises from failure of both ventilation and respiratory mechanics, posing a significant challenge in clinical settings. Type II respiratory failure can result from various underlying pathologies, including chronic obstructive pulmonary disease (COPD), obesity hypoventilation syndrome, neuromuscular disorders, and central respiratory depressant effects caused by medications or brain injuries. In patients with COPD, for instance, the progressive decline in pulmonary function and air trapping leads to ineffective ventilation, resulting in carbon dioxide retention. Similarly, individuals with obesity hypoventilation syndrome experience mechanical obstruction due to excessive body weight, which¡¦(»ý·«)
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